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Ⅱ型糖尿病骨质疏松和甲旁低 被引量:2

The Osteoporosis in NIDDM and Hypoparathyroidism
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摘要 选择Ⅱ型糖尿病患者60例(有骨质疏松者40例)和正常对照组21例,同时测定血甲状旁腺激素、血钙、血磷、碱性磷酸酶、血糖和骨密度。结果糖尿病组血碱性磷酸酶高于正常对照组,差异有显著性(t=2.23,P<0.05),骨密度明显降低,差异有极显著性(t=6.61,P<0.01),糖尿病有骨质疏松组较无骨质疏松组血甲状旁腺激素下降,差异有显著性(t=2.21,P<0.05),骨密度明显降低,差异有显著性(t=11.42,P<0.01),糖尿病组中血糖高者较血糖低者血甲状旁腺激素有下降趋势。因此,糖尿病继发骨质疏松时可呈甲旁低的表现。 Sixty cases of non-insulin-dependent diabetes mellitus (NIDDM )and 21 normal controls were selected, and their serum, calcium (Ca) .phosporus (P) , alkaline phosphatase( AKP) .parathyroid hormone(PTH) ,fast-ing blood sugar(FBS) levels and bone mineral densities(BMD) were measured. The results showed that AKP levels in NIDDM group were higher than the normals(t = 2. 23,P<0. 05);levels of BMD were srgnificantly lower than normals (t = 6. 61,P<0. 01);PTH levels in the osteoporosis group of NIDDM were lower than the non-osteoporosis group of N'IDDM(t=2. 21, P<0. 05). The BMD in the osteoporosis group of NIDDM were significantly lower than non-osteoporosis group of NIDDM(t= 11. 42,P<0. 01). The PTH in high FBS ones were lower than the low FBS patrent.s,but there were no relations between them. So hypoparathyroidism would secondarily osteoporosis happen in NIDDM.
出处 《天津医药》 CAS 1995年第10期593-595,共3页 Tianjin Medical Journal
关键词 糖尿病 II型 骨质疏松 甲状旁腺 机能减退 parathyroid hormone bone mineral densities alkline phosphatase osteoporosis type Ⅱdiabetes mellitus(NIDDM)
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  • 1王维力,天津医药,1981年,9期,707页

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