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人乳头瘤病毒、端粒酶、染色体异常与宫颈癌关系的研究进展 被引量:11

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作者 高宗侠 凌斌
出处 《中国临床保健杂志》 CAS 2007年第3期325-327,共3页 Chinese Journal of Clinical Healthcare
基金 安徽省科技攻关计划项目(06013124B)
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参考文献16

  • 1[1]Baseman JG,Koutsky LA.The epidemiology of human papillomavirus infections[J].J Clin Virol,2005,32Suppl 1:S16-S24.
  • 2[2]Munoz N,Bosch FX,De Sanjose S,et al.Epidemiologic classification of human papillomavirus types associated with cervical cancer[J].N Engl J Med,2003,348 (6):518-527.
  • 3[3]Lee D K,Kim BC,Kim IY,et al.The human papilloma virus E7 oncoprotein inhibits transforming growth factor-beta signaling by blocking binding of the Smad complex to its target sequence[J].J Biol Chem,2002,277 (4):38557-38564.
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  • 5[5]Mark RP,Trent H,Roger D P,et al.Selection of cervical keratinocytes containing integrated HPV16 associates with episome loss and an endogenous antiviral response[J].PNAS,2006,103 (10):3822-3827.
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  • 7[7]Gudleviciene Z,Didziapetriene J,Ramael M,et al.Human papillomavirus and p53 polymorphism in Lithuanian cervical cancer patients[J].Gynecol Oncol,2006 102 (3):530-533.
  • 8[8]Tsezou A,Oikonomou P,Kollia P,et al.The Role of Human Telomerase Catalytic Subunit mRNA Expression in Cervical Dysplasias[J].Biol Med,2005,230(4):263-270.
  • 9[9]Van Duin M,Steenbergen RD,De Wilde J,et al.Telomerase activity in high-grade cervical lesions is associated with allelic imbalance at 6Q14-22[J].Int J Cancer,2003,105:577-582.
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