摘要
新生儿高氧肺损伤是临床高氧治疗后较常见的并发症,目前尚无特异有效的防治方法.最近研究表明生长因子、骨髓间充质干细胞、促红细胞生成素、抗炎及抗氧化剂在新生儿高氧肺损伤的肺修复及保护中发挥了重要作用.该文综述以上分子系统在延缓和逆转新生儿高氧肺损伤病理过程中的作用机制.
Hyperoxia-induced lung injury in neonatal is a common complication after the clinical high oxygen therapy. There is not specific and effective prevention and treatment. Recent research suggests that many factor play an important role in the repair and protection of hyperoxia-induced lung injury in neonatal, such as growth factor, bone marrow mesenchymal stem cells,erythropoietin, anti-chemokine and anti-oxidation. The article reviews the molecular systems mechanisms in the pathologic process of hyperroxia-induced lung injury in neonatal.
出处
《国际儿科学杂志》
2010年第6期579-581,共3页
International Journal of Pediatrics
基金
四川省教育厅科研基金（08ZA150）
关键词
新生儿
高氧肺损伤
修复
Neonatal
Hyperoxia-induced lung injury
Repair
