1Howden CW. Clinical. Expression ofHelicobacter pylori infection. Am J Med 1996;100(Suppl 5A):27S
2Peek RM, Miller GG, Tham KT et al. Heightened inflammatory response and cytokineexpression in vivo to cagA+ Helicobacter Pylori strains. Lab Invest 1995; 73(6):760
3Owen RJ, Fugura N, Moreno M. Biotypes and DNA fingerprints of cytotoxigenicHelicobacter pylori from patients with gastritis and peptic ulcers and from patients withchronic gastritis only. J Clin Microbiol 1989; 27: 225
4Go MF, Chan,KY,Versalovid J, et al. Cluster analysis of Helicobacter pylori genomicDNA figerprints suggests gastroduodenal disease- specific associations. Scand JGastroenterol 1995 ; 30: 640
5Crabtree JE, Taylor JD, Wyatt JI, et al. Expression of 120 kilodalton protein andcytotoxincity in Helicobacter pylori. J Clin Pathol 1992 ;45: 733
6Kuipers EJ, Uyterlinde AM, Peina AS. et al. Longterm sequelae of Helicobacterpylori gastritis. Lancet 1995;345:1525
7Morales TG, Sampliner RE, Bhattacharyya A. Intestinal metaplasia of the gastriccardia. AM J Gastroenterol 1997;92(3):414
8Blaser MJ, Perez-perez GI, Kleanthous H. Infection with Helicobacter pylori strainspossessing cagA is associated with an increased risk of developing andenocarcinoma of thestomach. Cancer Res 1995;55:2111
9EL-Othar EM Oien K, EL-Nujumi, A et al. Helicobacter pylori infection and chronicgastric acid hyposecretion. Gastroenterology 1997 ; 113 ( 1 ): 15
10Feng J, Funk WD, Wang SS The RNA component of human telomerase. Science 1995;269j: 1236